Understanding Emotional Gravity In Relation to Cancer Research Part 3
The un coded DNA, much of which was previously dismissed as "junk," accounts for 99 percent of a cell's genome. A large number of mutations in cancer have been identified in the past several decades, but all have been found within the actual genetic blueprints for proteins. "This new finding represents an initial foray into the 'dark matter' of the cancer genome," said Levi Garraway, Harvard Medical School associate professor of medicine. These mutations affect a promoter region (a stretch of DNA code that regulates the expression of a gene) adjacent to the TERT gene. TERT contains the recipe for making telomerase reverse transcriptase, an enzyme that can make cells virtually immortal, and is often found over expressed in cancer cells. A promoter region of DNA controls the rate of a gene's transcription (the copying of its DNA recipe) into a message used by the cell to manufacture a protein.
Under normal conditions, cell reproduction is carefully controlled by the body. An important component of cell reproduction are telomeres, ('end tabs' on chromosomes). As normal cells divide, the telomeres gradually grow shorter until they become so short that the cell stops dividing and dies. This process relies on an enzyme called telomerase, which is sort of like a growth factor that temporarily maintains the length of the telomeres and enables the cell to continue reproducing.
Cancer, characterised by the uncontrolled growth of cells, rapidly divide and proliferate. The TERT promoter gene basically controls the instructions for making the telomerase enzyme. When this gene is mutated, the telomeres never shorten--which means the cells can divide and replicate forever. A mutation of the TERT promoter gene has been implicated in cancer.
Researchers from the Duke Cancer Institute analysed more than 1,200 tumours across 60 types of cancer and identified nine tumour types that are highly associated with mutations of the TERT promoter gene. These cancers share a common trait in that they originate in tissues with relatively low rates of cell renewal--indicating that they might require the mutation of the TERT promoter gene to trigger the abnormal production of telomerase. In contrast, the researchers found almost no TERT promoter mutations in many of the major cancer types, including breast and prostate cancer--indicating that an unknown factor is causing the telomeres to elongate and promote cell immortality in those diseases. However, they noted that the results in brain tumours were notable. In fact, this is the most frequent genetic mutation yet identified for primary glioblastoma, a deadly type of brain tumour.
The Oxford Journal of Gerontology published an article in 2011 investigating the connection between psychological stress, emotional health, and relative mean Telomere length in an ethnically homogeneous population of 4,441 women, aged 41-80 years. Mean Telemere length was measured using high-throughput quantitative real-time polymerase chain reaction. Social adversity exposure and emotional health were assessed through questionnaire and covariates through direct measurement and questionnaire. This study found evidence that adverse experiences during childhood may be associated with shorter Telomere length. This finding remained after covariate adjustment and showed evidence of a dose-response relationship with increasing number of reported childhood difficulties associated with decreasing relative mean Telomere length. No associations were observed for any of the other summary measures of social adversity and emotional health considered. These results extend and provide support for some previous findings concerning the association of adverse experience and emotional health histories with shorter Telemere length in adulthood.
In recent years, shorter telomeres have become associated with a broad range of aging-related diseases, including many forms of cancer, stroke, vascular dementia, cardiovascular disease, obesity, osteoporosis and diabetes. It can therefore be considered that chromosome mutation caused by the telomorase enzyme results from incoherent quantum entanglement, which, on the one hand causes a shortening of the coherent telomere gene and a lengthening of the incoherent telomorase.
It is now believed, as a result of a small pilot study by by UCFS, that a healthier lifestyle change can help grow the telomere gene. Changes in attitude, diet, exercising and toxic habits, can all help this extraordinary negentropic process to occur,
Our genes, and our telomeres, are not necessarily our fate," said lead author Dean Ornish, MD, UCSF clinical professor of medicine, and founder and president of the Preventive Medicine Research Institute. He stated, "So often people think 'Oh, I have bad genes, there's nothing I can do about it, But these findings indicate that telomeres may lengthen to the degree that people change how they live. Research indicates that longer telomeres are associated with fewer illnesses and longer life."
Longer, healthier telomeres helps generate coherent asymmetrical emotional quantum entanglement mitosis, which lessens the chance of mutations in the genetic sequence. This indicates that a coherent emotional field is the key to better health and longevity?
The new study by UCSF was designed to determine if the lifestyle changes would affect telomere length and telomerase activity in men in the early stages of prostate cancer, over a long time period. "This was a breakthrough finding that needs to be confirmed by larger studies," said co-senior author Peter R. Carroll, MD, MPH, professor and chair of the UCSF Department of Urology. "Telomere shortening increases the risk of a wide variety of chronic diseases," Carroll said. "We believe that increases in telomere length may help to prevent these conditions and perhaps even lengthen lifespan."
Stuart Hameroff points out that current therapies for cancer are generally aimed at impairing mitosis and are thus severely toxic. Many cancer drugs bind to microtubules and prevent their disassembly/assembly required for formation and activities of the mitotic spindles. In addition to generalised toxicity due to impairment of non-mitotic microtubule function, partial disruption of mitosis can cause further incoherence in the emotional quantum field. Radiation is also a toxic process with the goal of impairing/destroying highly active malignant cells more than normal cells. Recognizing centrosomes as the keys organising factor in mitosis, proposed disabling centrosomes by cooling/freezing is becoming a considered cancer therapy.
Low level laser illumination apparently enhances mitosis. As it enhances centriole replication it has the opposite of the desired effect in cancer therapy. On the other hand if centrioles are sensitive to coherent light, then higher intensity laser illumination (still below heating threshold) may selectively target centrioles, impair mitosis and be a beneficial therapy against malignancy. However laser illumination may also be used in a more elegant mode. If centrioles utilize quantum photons for entanglement, properties of centrosomes/centrioles approached more specifically could be useful for therapy.
Healthy centrioles for a given organism or tissue differentiation should then have specific quantum optical properties detectable through some type of readout technology. An afflicted patient's normal cells could be examined to determine the required centriole properties which may then be used to generate identical quantum coherent photons administered to the malignancy. In this mode the idea would not be to destroy the tumour (relatively low energy lasers would be used) but to "reprogram" or re-differentiate the centrioles and transform the tumour back to healthy well differentiated tissue.
It is suggested here that normal mitosis is organized by emotional quantum entanglement and quantum coherence among centrioles. In particular, quantum optical properties of centrioles enable emotional entanglement in normal mitosis, which ensures precise mirror-like activities of mitotic spindles and daughter chromatids, and proper differentiation, communication and boundary recognition between daughter cells.
Defects in the proposed mitotic quantum entanglement/coherence can explain all aspects of malignancy. Analysis and duplication of quantum optical properties of normal cell centrioles could possibly lead to laser-mediated therapeutic disruption and/or reprogramming of cancerous tumours as well as abundant, ethical production of stem cells. This finding suggests that cells with shortened Telomere length become the target cells for cancer - the Telomere length, having been affected and possibly caused by negative emotions. They become vulnerable and open to invasion. Here we have our direct link between Cancer, Gravity and emotion.
Read more: http://lifestyle.inquirer.net/122139/is-cancer-caused-by-negative-emotions#ixzz33daZSei5
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Under normal conditions, cell reproduction is carefully controlled by the body. An important component of cell reproduction are telomeres, ('end tabs' on chromosomes). As normal cells divide, the telomeres gradually grow shorter until they become so short that the cell stops dividing and dies. This process relies on an enzyme called telomerase, which is sort of like a growth factor that temporarily maintains the length of the telomeres and enables the cell to continue reproducing.
Cancer, characterised by the uncontrolled growth of cells, rapidly divide and proliferate. The TERT promoter gene basically controls the instructions for making the telomerase enzyme. When this gene is mutated, the telomeres never shorten--which means the cells can divide and replicate forever. A mutation of the TERT promoter gene has been implicated in cancer.
Researchers from the Duke Cancer Institute analysed more than 1,200 tumours across 60 types of cancer and identified nine tumour types that are highly associated with mutations of the TERT promoter gene. These cancers share a common trait in that they originate in tissues with relatively low rates of cell renewal--indicating that they might require the mutation of the TERT promoter gene to trigger the abnormal production of telomerase. In contrast, the researchers found almost no TERT promoter mutations in many of the major cancer types, including breast and prostate cancer--indicating that an unknown factor is causing the telomeres to elongate and promote cell immortality in those diseases. However, they noted that the results in brain tumours were notable. In fact, this is the most frequent genetic mutation yet identified for primary glioblastoma, a deadly type of brain tumour.
The Oxford Journal of Gerontology published an article in 2011 investigating the connection between psychological stress, emotional health, and relative mean Telomere length in an ethnically homogeneous population of 4,441 women, aged 41-80 years. Mean Telemere length was measured using high-throughput quantitative real-time polymerase chain reaction. Social adversity exposure and emotional health were assessed through questionnaire and covariates through direct measurement and questionnaire. This study found evidence that adverse experiences during childhood may be associated with shorter Telomere length. This finding remained after covariate adjustment and showed evidence of a dose-response relationship with increasing number of reported childhood difficulties associated with decreasing relative mean Telomere length. No associations were observed for any of the other summary measures of social adversity and emotional health considered. These results extend and provide support for some previous findings concerning the association of adverse experience and emotional health histories with shorter Telemere length in adulthood.
In recent years, shorter telomeres have become associated with a broad range of aging-related diseases, including many forms of cancer, stroke, vascular dementia, cardiovascular disease, obesity, osteoporosis and diabetes. It can therefore be considered that chromosome mutation caused by the telomorase enzyme results from incoherent quantum entanglement, which, on the one hand causes a shortening of the coherent telomere gene and a lengthening of the incoherent telomorase.
It is now believed, as a result of a small pilot study by by UCFS, that a healthier lifestyle change can help grow the telomere gene. Changes in attitude, diet, exercising and toxic habits, can all help this extraordinary negentropic process to occur,
Our genes, and our telomeres, are not necessarily our fate," said lead author Dean Ornish, MD, UCSF clinical professor of medicine, and founder and president of the Preventive Medicine Research Institute. He stated, "So often people think 'Oh, I have bad genes, there's nothing I can do about it, But these findings indicate that telomeres may lengthen to the degree that people change how they live. Research indicates that longer telomeres are associated with fewer illnesses and longer life."
Longer, healthier telomeres helps generate coherent asymmetrical emotional quantum entanglement mitosis, which lessens the chance of mutations in the genetic sequence. This indicates that a coherent emotional field is the key to better health and longevity?
The new study by UCSF was designed to determine if the lifestyle changes would affect telomere length and telomerase activity in men in the early stages of prostate cancer, over a long time period. "This was a breakthrough finding that needs to be confirmed by larger studies," said co-senior author Peter R. Carroll, MD, MPH, professor and chair of the UCSF Department of Urology. "Telomere shortening increases the risk of a wide variety of chronic diseases," Carroll said. "We believe that increases in telomere length may help to prevent these conditions and perhaps even lengthen lifespan."
Stuart Hameroff points out that current therapies for cancer are generally aimed at impairing mitosis and are thus severely toxic. Many cancer drugs bind to microtubules and prevent their disassembly/assembly required for formation and activities of the mitotic spindles. In addition to generalised toxicity due to impairment of non-mitotic microtubule function, partial disruption of mitosis can cause further incoherence in the emotional quantum field. Radiation is also a toxic process with the goal of impairing/destroying highly active malignant cells more than normal cells. Recognizing centrosomes as the keys organising factor in mitosis, proposed disabling centrosomes by cooling/freezing is becoming a considered cancer therapy.
Low level laser illumination apparently enhances mitosis. As it enhances centriole replication it has the opposite of the desired effect in cancer therapy. On the other hand if centrioles are sensitive to coherent light, then higher intensity laser illumination (still below heating threshold) may selectively target centrioles, impair mitosis and be a beneficial therapy against malignancy. However laser illumination may also be used in a more elegant mode. If centrioles utilize quantum photons for entanglement, properties of centrosomes/centrioles approached more specifically could be useful for therapy.
Healthy centrioles for a given organism or tissue differentiation should then have specific quantum optical properties detectable through some type of readout technology. An afflicted patient's normal cells could be examined to determine the required centriole properties which may then be used to generate identical quantum coherent photons administered to the malignancy. In this mode the idea would not be to destroy the tumour (relatively low energy lasers would be used) but to "reprogram" or re-differentiate the centrioles and transform the tumour back to healthy well differentiated tissue.
It is suggested here that normal mitosis is organized by emotional quantum entanglement and quantum coherence among centrioles. In particular, quantum optical properties of centrioles enable emotional entanglement in normal mitosis, which ensures precise mirror-like activities of mitotic spindles and daughter chromatids, and proper differentiation, communication and boundary recognition between daughter cells.
Defects in the proposed mitotic quantum entanglement/coherence can explain all aspects of malignancy. Analysis and duplication of quantum optical properties of normal cell centrioles could possibly lead to laser-mediated therapeutic disruption and/or reprogramming of cancerous tumours as well as abundant, ethical production of stem cells. This finding suggests that cells with shortened Telomere length become the target cells for cancer - the Telomere length, having been affected and possibly caused by negative emotions. They become vulnerable and open to invasion. Here we have our direct link between Cancer, Gravity and emotion.
Read more: http://lifestyle.inquirer.net/122139/is-cancer-caused-by-negative-emotions#ixzz33daZSei5
Follow us: @inquirerdotnet on Twitter | inquirerdotnet on Facebook