Gout is an inflammatory disease that predominately affects the joints, tendons and other bodily tissues.
Closely related to rheumatic arthritis, the medical condition is more common in males, yet it is also known to develop in post- menopausal women and hypertensive people.
There is no permanent cure for gout which suggests that gout therapy is required to control the progression of the condition.
A gout attack occurs suddenly during the night and is extremely painful.
The symptoms of the medical condition are also sudden, yet dominant.
When the joint is affected, the pain may last for long periods of time; days or weeks and reoccur months or years later.
A chronic gout attack has the potential to induce permanent organ and tissue damage.
A gout patient generally experiences redness, swelling, tenderness, tightness warmth and skin lump in the affected area.
In most cases, initial gout attacks occur in the big toe.
The goals for gout therapy are comprehensive in that they are implemented to end the initial pain and deterring the possibility of future attacks thereafter.
Gout is caused by hyperuricemia; a buildup of uric acid in the bloodstream.
It is the result of an imbalance in the production and excretion of urate.
Too much uric acid alleviates the kidneys' ability to flush away excess toxins.
Excess production of uric acid is known to be caused by the intake of large quantities of the chemical called purine.
Also, excessive consumption of fructose can cause uric acid buildup, although not as common.
The buildup forms hard crystals that deposits on the joints; thereby causing an infection.
There are several risk factors that increase the chance of developing gout.
These risk factors include being overweight or obese, having hypertension, excessive alcohol intake, diuretic use, and a diet rich in meats and seafood.
Meats and seafood is highly rich in purines.
The ultimate goal of treatment is to ease the initial pain and reduce uric acid buildup in order to prevent the formation of tophi and kidney stones (chronic gout).
Gout is treated with nonsteroidal anti-inflammatory drugs, steroids and colchicine.
Changing medications associated with high blood pressure such as diuretics may help reduce the potential for future gout attacks.
Essential life- style modifications include a diet that is low in purine intake.
You must also loose weight and decrease alcohol consumption.
Gout therapy is designed to assist the patient with avoiding anything that will trigger the onset of the symptoms.
Closely related to rheumatic arthritis, the medical condition is more common in males, yet it is also known to develop in post- menopausal women and hypertensive people.
There is no permanent cure for gout which suggests that gout therapy is required to control the progression of the condition.
A gout attack occurs suddenly during the night and is extremely painful.
The symptoms of the medical condition are also sudden, yet dominant.
When the joint is affected, the pain may last for long periods of time; days or weeks and reoccur months or years later.
A chronic gout attack has the potential to induce permanent organ and tissue damage.
A gout patient generally experiences redness, swelling, tenderness, tightness warmth and skin lump in the affected area.
In most cases, initial gout attacks occur in the big toe.
The goals for gout therapy are comprehensive in that they are implemented to end the initial pain and deterring the possibility of future attacks thereafter.
Gout is caused by hyperuricemia; a buildup of uric acid in the bloodstream.
It is the result of an imbalance in the production and excretion of urate.
Too much uric acid alleviates the kidneys' ability to flush away excess toxins.
Excess production of uric acid is known to be caused by the intake of large quantities of the chemical called purine.
Also, excessive consumption of fructose can cause uric acid buildup, although not as common.
The buildup forms hard crystals that deposits on the joints; thereby causing an infection.
There are several risk factors that increase the chance of developing gout.
These risk factors include being overweight or obese, having hypertension, excessive alcohol intake, diuretic use, and a diet rich in meats and seafood.
Meats and seafood is highly rich in purines.
The ultimate goal of treatment is to ease the initial pain and reduce uric acid buildup in order to prevent the formation of tophi and kidney stones (chronic gout).
Gout is treated with nonsteroidal anti-inflammatory drugs, steroids and colchicine.
Changing medications associated with high blood pressure such as diuretics may help reduce the potential for future gout attacks.
Essential life- style modifications include a diet that is low in purine intake.
You must also loose weight and decrease alcohol consumption.
Gout therapy is designed to assist the patient with avoiding anything that will trigger the onset of the symptoms.